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Cancer and Vitamin B17 Laetrile, click here

For nearly thirty years the media blasted you with scare stories about the "deadly" cyanide content in Vitamin B17 Laetrile, warning that anyone daring to eat apricot kernals would fall dead on the floor in seconds. This is simply not true, and B17 remains one of the most potent controls for cancer anywhere in the world. Recently medical researchers in London "discovered" that the cyanide found in B17 can kill tumours, though of course no-one dared mention apricots. The researchers used cyanide derived from the Cassava root instead, which as you will see here contains exactly the same active cyanide ingredients as apricots!

IS CANCER MERELY A VITAMIN DEFICIENCY DISEASE?
Copyright Joe Vialls, edited 20 Jan 2001, All Rights reserved

         Though many alternative treatments for cancer have demonstrated success over the years, there has been too little discussion on possible ways of preventing or suppressing cancer before it has a chance to attack, or controlling it effectively after it strikes. There is also the question of why we have become increasingly vulnerable to every form of cancer as each year goes by. Is there a direct link between easily-researched and steadily increasing quantities of chemicals and additives in our food and water supplies, or has the removal of certain intrinsic factors from our refined western diets been more to blame?  Specifically where vulnerability is concerned, there is compelling evidence that the removal of vitamin B17 from our diets has played the greatest single role.
         Decades ago two books provided vital clues to one possible cause for the alarming increasing incidence of all forms of cancer. Though aimed at widely different readerships, both books looked at possible connections  between vitamin deficiency and disease.  "World Without Cancer" by Edward Griffin of California examines the growing conviction that vitamin B17 appears capable of preventing the onset of all forms of cancer, while the second book "Eat Fat and Grow Slim" by Richard MacKarness M.D., proposes a high fat diet for those tired of trying the 'lettuce leaf' approach to weight loss.
         Initially it is difficult to see any connection between these two widely differing subjects, but connection there is. In their individual ways both books point towards two entirely different groups of people (one vegetarian and the other carnivorous) who suffer no cancers or heart disease at all while consuming local food in their native environments. This is a critical point, for without such a linkage it is far too easy for established medical researchers to lead the public astray with billions of dollars worth of research aimed at promoting radiation and chemotherapy treatments. During 1950 after many years of research a dedicated biochemist by the name of Dr. Ernst T. Krebs, Jr. isolated a new vitamin that he numbered B17 and called 'Laetrile'. As the years rolled by, thousands became convinced that Krebs had finally found the complete control for all cancers, a conviction shared by even more people today. Back in 1950 Ernst Krebs had no idea of the hornet's nest he was about to stir up.
         Unable to patent B17 or claim exclusive rights to the vitamin, the pharmaceutical multinationals launched a massive propaganda attack of unprecedented viciousness against Laetrile, despite the fact that hard proof of its efficiency in controlling cancer now surrounds us. How is it any of us gets cancer in the first place - through exposure to cigarette smoking, intense sunlight or perhaps the effect of toxic food additives? Dr. Krebs thinks not. All of his hard biochemical evidence points to the fact that cancer is a simple deficiency disease of vitamin B17, long ago removed from our highly refined western diets. Krebs postulates that the so-called 'carcinogens' are merely stress triggers which  finally expose the B17 deficiency with devastating effect.  The credibility of Krebs' claim is best illustrated by the vitamin C deficiency disease known as 'scurvy'. As with cancer there is no advance warning of scurvy; no tell-tale signs that the body is running low on vitamin C reserves. One minute the patient is a healthy person and the next an invalid. Recovery from scurvy is equally dramatic. Within days (sometimes hours) of high-dose vitamin C treatment the scurvy vanishes, reappearing only if vitamin C reserves once more drop below a critical (but undocumented) level.  Thus if Ernst Krebs is right, alternative treatments such as Essiac, Oxygen, and electromagnetic therapies are continually fighting an uphill battle. The simple replacement of the 'lost' Vitamin B17 in our diets might contribute to an increased success rate for other alternative treatments, or replace them completely.
         The proof Dr. Krebs has presented over the years to support his claim of 'lost' B17 in our diets is impressive. Centuries ago we used to eat millet and linseed bread rich in B17, but now we chew our way through wheat bread which has none at all. For generations our grandmothers used to carefully crush the seeds of plums, greengages, cherries, apples, apricots and other members of the botanical family Rosaceae, and diligently mix the kernels with their home made jams and preserves.  Grandma probably didn't know why she was doing it, but the kernels of all these fruits are some of the most potent sources of B17 in the world.  In the tropics, huge quantities of B17 are found in bitter cassava, also known as tropical manioc. Research has proved that a Himalayan tribe known as the 'Hunza' never contract cancer or suffer from heart disease if they stick to their native diet which is exceptionally high in both apricots and millet. However, once exposed to western diets the Hunza become as vulnerable as the rest of us. Because the  Hunza eat very little meat this might encourage large numbers of the vegetarian community to pat each other eagerly on the back whilst proclaiming: "See, we were right!"
         Alas, such jubilation would be sadly misplaced.  In a bid to find a diet acceptable to those not wishing to nibble one lettuce leaf a day, Richard MacKarness made a detailed study of Eskimos living on the polar ice, and American Indians eating traditional diets.  In their natural environments both groups are mostly carnivorous, eating wild game including Elk and Caribou, supplemented only by  wild berries when available in season. The main point MacKarness makes in his book "Eat Fat and Grow Slim" is that there is no such thing as obesity among these people: an interesting fact in its own right as they regularly gorge themselves on saturated animal fats at least twice a day. Where things get decidedly more interesting is his proof that Eskimos and Indians living in their natural environments and eating traditional foods, NEVER contract cancer or suffer from heart  complaints: exactly the same as the Hunza people in the Himalayas, despite the Eskimos and American Indians being carnivores rather than vegetarians. Careful investigation reveals the most likely common factor to be vitamin B17. The caribou which form a large part of the staple diet of both groups graze predominantly on arrow grass containing up to 15,000 mg per kilo nitriloside, the primary source of B17.  The salmon berries dried and eaten by Eskimos and Indians alike also contain huge quantities of vitamin B17. So in these widely differing communities vegetarians and carnivores alike can both remain perfectly healthy. This is of particular importance to those who are environmentally unable to take up a vegetarian diet by choice.  Such a diet would be well nigh impossible on the polar ice caps or in arid deserts.
         Unfortunately for most 'civilized' western cultures, grasses and other foodstuffs now used to feed domestic animals intended for human consumption rarely contain more than a trace of nitriloside, though they did until botanists and biochemists started  to genetically alter our plant life. In turn this means our secondary source of vitamin B17 (through the meat food-chain) is fast drying up. Where The Hunza or Eskimaux get an average individual ration of between 250 and 3,000 milligrams of vitamin B17 every day, European folk eating 'healthy' modern foods receive barely 2 milligrams.  The implications of these finding are staggering of course. If we managed to control scurvy centuries ago, how is it we cannot do the same for cancer today?  The fact is we probably could if our respective governments would allow it.  Unfortunately most: governments have buckled under the pressure exerted by the pharmaceutical multinationals, the American Food & Drug Administration, and the American Medical Association. All three have mounted highly successful 'scare' campaigns based on the fact that vitamin B17 contains quantities of 'deadly' cyanide; conveniently forgetting that vitamin B12 also contains large quantities of cyanide but is freely available in health food shops world-wide.
         Dr. Kreb's B17 Laetrile was derived from apricot kernels and then synthesised into crystalline form using his own unique process.  Suddenly the American FDA bombarded the media with a  story about an unfortunate couple who had poisoned themselves by eating raw apricot kernels in San Francisco. The story made headline news across the U.S.A. although several suspicious ;journalists never managed to establish the identity of the unfortunate couple, despite many determined attempts.  But the multinational pharmaceutical/FDA boot had been put in with a vengeance. From that point onwards eating apricot kernels or B17 Laetrile became synonymous with committing suicide. Back in the fifties Dr. Ernst Krebs proved beyond doubt that B17 was completely harmless to humans in the most convincing way possible. After testing the vitamin on animals, he filled a large hypodermic with a mega-dose of concentrated Laetrile which he then injected into his own arm! Drastic perhaps, but the adventurous Dr. Krebs is still alive and well today.
         The vitamin is harmless to healthy tissue for a very simple  reason:  each molecule of B17 contains one unit of cyanide, one unit of benzaldehyde and two of glucose (sugar) tightly locked together. In order for the cyanide to become dangerous it is first necessary to 'unlock' the molecule to release it, a trick that can only be performed by an enzyme called beta-glucosidase, which is present all over the human body in minute quantities, but in truly vast quantities (up to 100 times as high) at only one place: the site of a malignant cancer tumour. Thus the cyanide is unlocked only at the cancer site with drastic results, which become utterly devastating to the cancer cells because the benzaldehyde unit unlocks at the same time.  Benzaldehyde is a deadly poison in its own right, which then acts synergistically with the cyanide to produce a poison 100 times more deadly than either in isolation. The combined effect on the cancer cells is best left to the imagination. But what about danger to the rest of the body's cells? Another enzyme, rhodanese, always present in far larger quantities than the unlocking enzyme beta-glucosidase in healthy tissues, has the easy ability to completely break down both cyanide and benzaldehyde into beneficial body products. Predictably perhaps, malignant cancer cells contain no rhodanese at all, leaving them completely at the mercy of the two deadly poisons.
         Generations ago our agricultural experts knew of the 'trigger' effect of beta-glucosidase i.e. its ability to unlock the cyanide unit in the B17 molecule, but there appeared to be a considerable amount of confusion about how to approach the problem. The simplistic solution seemed to be that of labeling all plants containing the B17 molecule "poisonous", then genetically modifying them to remove the nitriloside content completely for the safety of the animals. One classic example of this misguided approach was a 1940s case where Australian sheep were occasionally dying from an excess of cyanide derived from white clover, known to contain B17. Without giving a thought to why most of the sheep eating the same clover stayed alive,  botanists promptly bred the nitriloside content out of the white clover. In reality the sheep that died were the few who wandered away from the clover to eat a tasty fuschia plant which contained a very high concentration of the unlocking enzyme beta-glucosidase, which reacted immediately in the sheeps' stomachs and caused death. If the botanists had neutralised a few fuschias instead of millions of tons of white clover, there would be significantly more vitamin B17 available today for humans to ingest through the meat food-chain.
         For better or worse vast quantities of vitamin B17 have been removed from western foods, and society is now faced with cancers at an unprecedented level. Even if we allow that a deficiency of B17 might be the most likely culprit for the sudden appearance of such a condition, there is still the question of what happens next and how that cancer develops to the life-threatening stage. In "World Without Cancer", Griffin explains the trophoblastic theory of cancer proposed by Professor John Beard of Edinburgh University, who claims certain pre-embryonic cells in pregnancy differ in no discernible way from highly malignant cancer cells. Griffin notes: ' The  trophoblast  in pregnancy  indeed  does  exhibit  all the   classical characteristics of cancer. It spreads and multiplies rapidly as it eats its way into the uterus wall preparing a   place where the embryo can attach itself for maternal protection and nourishment.' 
         The trophoblast is formed in a chain reaction by another cell which Griffin simplifies down to the 'total-life' cell, which can evolve into any organ or tissue, or alternatively into a  complete human embryo. When the total-life  cell is triggered into producing trophoblast by contact with the hormone oestrogen, present in both males and females, one of two different things happens: in the case of pregnancy the result is conventional development of a placenta and umbilical cord. If the trophoblast is triggered as part of a healing process however, the result is cancer or, as Edward Griffin cautions: 'To be more accurate, we should say it is cancer if the healing process is not terminated upon completion of its task.' Stunning proof of this claim is readily available. All trophoblast cells produce a unique hormone called the chorionic gonadotrophic (CGH) which is easily detected in urine. Thus if a person is either pregnant or has cancer, a simple CGH pregnancy test should confirm either or both. It does, with a reported accuracy of better than 85%. If the urine sample shows positive it means either normal pregnancy or abnormal malignant cancer. Griffin notes: 'If the patient is a woman, she either is pregnant or has cancer. If he is a man, cancer can be the only cause.'  So why all of the expensive, dangerous biopsies carried to 'detect' cancerous growths? One can only assume that medicare pays doctors a larger fee for biopsies than pregnancy tests. In Australia, two CGH style 'do-it-yourself' pregnancy tests stocked by most pharmacies are 'Discover' and 'Predictor'.
         Physicians reading this article will probably be shaking with self-righteous indignation by this stage, muttering darkly: 'Yes, but where is the PROOF?' Here: Most people have heard of 'spontaneous regression' where  a cancer simply goes away, hopefully never to reappear. Such spontaneous regressions are exceedingly rare and vary from one form of cancer to another. One virulent cancer variety known as testicular  chorionepithelioma has never been known to produce a single spontaneous regression. Perhaps for that precise reason, Dr.  Krebs singled it out for special attention when proving the effectiveness of B17 Laetrile in providing total control for cancers.
         As Edward Griffin recalls, in a banquet speech in San Francisco on November 19, 1967, Dr.  Ernst T. Krebs briefly reviewed six cases of testicular chorionepithelioma.  Then he added: 'Now there is an advantage in not having had prior radiation, because if you have not received prior radiation that has failed, then you cannot enjoy the imagined benefits of the delayed effects of prior radiation. So this boy falls into the category of the "spontaneous regression." And when we look at this scientifically, we know that spontaneous regression occurs in fewer than one in 150,000 cases of cancer. The statistical possibility of spontaneous regression accounting for the complete resolution of six successive cases of testicular chorionepithelioma [All six completely resolved solely by B17 Laetrile - Ed.] is far greater than the statistical improbability of the sun not rising tomorrow morning. '
         Wisely perhaps, Griffin notes that because of the adverse publicity against B17 Laetrile, and because of the difficulties in obtaining the 'banned' substance, most cancer sufferers turn to the vitamin as a last resort, long after they have been burned by radiation therapy, and/or poisoned by chemotherapy.
         When "World Without Cancer" was written in 1974, B17 Laetrile  was freely available in Australia. It is not now. A recent check with the Australian Cancer Foundation and health authorities revealed that nowadays Canberra considers each individual case on its merits, then decides whether the patient should be allowed to import sufficient of the material for his or her own personal use. If he or she manages to jump that hurdle, it is then his or her own responsibility to find a doctor prepared to inject it.  Seemingly the multinational lobbyists managed to get to our politicians before  Dr. Krebs could get to the Australian public.
         Last month Australian nationwide television carried the frightening news that two out of every three Australians can expect to suffer skin cancer at least once during their lifetimes. On the massive evidence provided by Dr. Ernst Krebs, Jr., Edward Griffin and Dr. Richard MacKarness, that figure might be crushed to a tiny percentage if Australians were allowed freedom of choice where B17 Laetrile is concerned.  It is perhaps time for Australians to take a stand on this undeniably lethal issue.


REFERENCES


Australian Veterinary Journal, Franklin and Reid, Volume 100, p92, 1944.


CANCER: Disease of Civilization? An Anthropological and Historical Study, Stefanson, V, Hill & Wang, New York, 1960.


Eat Fat and Grow Slim, MacKarness, R., Fontana, London 1976.


Laetrile Case Histories, Richardson J.A. and P, Bantam, USA.


New Zealand Journal of Science and Technology, Coop & Blakely, February 1949, page 277,  February 1950, page 45.


The Nitrilosides in Plants and Animals, "Nutritional and therapeutic Implications", Krebs E.T, Cancer Book House, Los Angeles, California, USA.  Published by the Cancer Control Society, a non-profit educational Society dedicated to educating the public about nutrition and non-toxic cancer therapies.


World Without Cancer, Griffin, G.E., American Media, PO Box 4646, Westlake Village, California 91359, USA. Price US$9.95 plus air mail postage of US$9.00. Discounts available (up to 65% for bulk orders) for alternative booksellers.


Footnote 1997


         For anyone unable to access synthesised Laetrile in Mexico, the most obvious source of oral vitamin B17 is apricot kernels. Unfortunately the Australian Government has made access to this natural product extremely difficult. Courtesy of the pharmaceutical lobbies, it is now a federal "crime" for health food shops and others to sell apricot kernels to the public. There is an easier way of obtaining B17 not shown in the text, and that is from crushed linseed. The primary source of B17 in linseed is Linimarin rather than Nitriloside found in apricot kernels and Cassava, but the effect is the same if enough is eaten. 
         Readers should note that linimarin B17 exists in the crushed linseed itself, not in the extracted linseed oil.  Cheap "Linseed Cake" sold by all animal feed stores is ideal, though in every case the linseed should be mixed in a high-speed blender or coffee grinder before use. Whole linseed is very hard, passing through the gut unaltered and without releasing its beneficial B17 content into the bloodstream. Once crushed, the linseed meal may be mixed with breakfast cereals or baked in bread.
         This report was first published in "Conscious Living" magazine, issue 27, February 1995, and has since been posted on a number of different internet sites.



"SETTING UP" LIBYA FOR THE LOCKERBIE BOMBING"
Copyright Joe Vialls, January 2000


         Most media hype alleging the two Libyans currently imprisoned in Holland are guilty of the bombing of Pan Am 103 over Lockerbie, can be traced back to a single book. Co-authored in 1993 by Donald Goddard and Lester Coleman III, "Trail Of The Octopus" spawned thousands of stories in the mainstream media. Indeed, without "Trail Of The Octopus" as a guide, it is difficult to see how the media could have generated any meaningful stories at all about Libyan "guilt" over the Lockerbie bombing. At the surface level the book deftly tries to shift part of the blame backs towards Israel's prime enemy Syria, thus embroiling yet another Arab nation in a sea of deceit and half truths. Whoever blew Pan Am 103 up over Lockerbie, the book all but shouts at the reader: "The culprits were not America or the State of Israel". 
         In essence the text is a series of recollections by Lester Coleman III, ghost written and edited by journalist Donald Goddard. Coleman claims to have been a consultant for the American Defense Intelligence Agency (DIA) with strong links to the Drug Enforcement Agency (DEA). If his claims are true, this would of course put Lester Coleman III in the driving seat on matters related to the Middle East, and in particular the bombing of Pan Am 103. But are his claims really true? The media made the fatal mistake of assuming they are, thereby setting up a huge house of cards ready for instant destruction by anyone sceptical enough to ask serious questions. If Lester Coleman III's claims can be proven untrue, then by default every one of the thousands of media stories which followed about Lockerbie are also untrue.
         This report is not about minor grammatical errors made by Goddard, or marginal recall about the spelling of an obscure contact's name by Coleman. It is about proven errors so large you could drive a 10-ton Mack truck through each and every one of them with room to spare.
         By his own account Coleman first arrived in Beirut for the DIA on 25 February 1985. At that time he claims part of his job was to liaise with, and provide intelligence for, the Green Berets [special forces] in place for a hostage rescue mission. In the words of Donald Goddard on p 144: " the main duties of the Asmar cell had been to monitor the Muslim radical groups supported by Iran and Syria, to report on the movements of their leaders - in particular, those of Sheikh Mohammed Hussein Fadellah - and keep track of the Western hostages then being taken in Beirut. After Coleman's arrival as Asmar's control and his communications link with DIA headquarters, this work continued "
         So Coleman specifically claims through Goddard in the book that he [Coleman] had a direct controlling role in the Beirut hostage drama from 25 February 1985. Later in the text he expands on this theme by mentioning hostage names such as Levin, Anderson, Keenan, McCarthy and Waite, but erroneously insists there was no rush about freeing the hostages at that time. Indeed, on pp 182-183 he claims: "We could have gotten the hostages out any damn time we wanted to, but nobody was willing to rock the boat with a rescue operation. We knew where they were. We knew who their guards were. We knew what they had for lunch. We knew when and where they were going to be moved before their guards did. But the DIA wouldn't risk any action based on information that might have been traced back to one of Asmar's people."
         This would have come as an enormous shock during early 1985 to no less a personage than William Casey, Director of the CIA, who at the time had a close working relationship with the heads of both the DIA and DEA. On 16 March 1984 his own CIA head of station in Beirut, William Buckley, was captured and became by far the most important hostage from the American Intelligence Community's point of view. Hizbollah was holding a senior member of the CIA hostage, and [according to Casey] slowly torturing him to death during the exact period Coleman claims that "We could have gotten the hostages out any damn time we wanted to".
         Unfortunately Coleman proves himself ignorant of this pivotal fact, and demonstrates he has no knowledge of the swarms of spooks criss-crossing Beirut, trying just about anything to get Buckley out of there during early 1985. Buckley's name is not mentioned anywhere in the text or index of "Trail of the Octopus" at all. Coleman proves he is also unaware that the Green Berets he "had to liaise with, and provide intelligence for", were in Beirut at that time primarily to try a snatch job on William Buckley.
         According to David Jacobsen, director of the city's American University Hospital, who was taken hostage some weeks before and was sharing a cell with Buckley, the latter finally died from a combination of abuse and pneumonia on his 444th day of captivity, during the evening of 3 June 1985. Hizbollah did not announce Buckley's death until 11 October 1985, but no matter which of the two dates is correct, the period January - June 1985 in Beirut was frantic, because Casey had pulled out all the stops and ordered a rescue mission regardless of cost.
         Predictably perhaps, Jacobsen also appears to have slipped Coleman's memory. Just like William Buckley's, his name is also missing from "Trail Of The Octopus". Because of Buckley's profile, and Casey's directive as Director of Central Intelligence (DCI), it is ridiculous to suggest that Coleman could "forget" to include this critical information in "Trail of the Octopus".
         Over the years I have attempted to investigate this staggering erroneous claim as best I could, because if Coleman was not performing the duties he claimed in Beirut during the time frame claimed in the book, then most of his other claims made in "Trail Of The Octopus" can reasonably be regarded as untrue and deliberately fabricated disinformation.
          Not one of the personnel I have so far contacted at Fort Bragg, North Carolina, remembers liaising with or receiving intelligence from, Lester Coleman III or his alias "Thomas Leavy" in Beirut.
         In late October 1996, Lester Coleman III was badly beaten by four masked men at his hideout in Spain. On arrival back on America on 17 October 1996 in a wheelchair, Coleman was arrested by the U.S. federal authorities, and charged with passport fraud. Hardly the treatment one would expect to be meted out by federal authorities to a bona-fide Defense Intelligence Agency consultant.

PSYCHOLOGICALLY ENGINEERING THE LOCKERBIE TRIAL

         On 15 August 1999 in a story titled "Airlines Fear Bomb Attacks", Murdoch Group newspapers suggested Islamic fanatics are planning another Lockerbie-style bombing sometime during the next three months. It is far more likely that those really responsible for bombing Pan Am 103 are planning a lookalike atrocity, designed to act as a psychological reinforcer during the trial of the two Libyan suspects in late 2000. There is no credible scientific evidence linking these two men to Pan Am Flight 103 or to the village of Lockerbie, but the American Department of State and the Israeli Mossad are growing increasingly desperate for a conviction.
                   In a nutshell the forged evidence against the two Libyans is as follows: The suspects took delivery in Malta of a bomb disguised as a Toshiba Bombeat portable radio, said to contain 312 grams of Semtex-H explosive coupled to a barometric switch and a timer. The suspects allegedly wrapped the bomb inside children's clothes bought in a Malta store, then packed it carefully inside a brown Samsonite suitcase for transit to Pan Am 103. The CIA claims the suitcase was then loaded by the suspects onto Air Malta Flight 180 to Frankfurt, where it was transhipped to a Pan Am 727 feeder flight to London Airport Heathrow. There it was allegedly transhipped again onto "Maid Of The Seas", the 747 which then obligingly exploded overhead Deans Cross in the Lake District, its 500 m.p.h. forward speed and inertia ensuring the debris and bodies finally fell to earth many miles further north at Lockerbie in Scotland.
         As will be explained later, it is a scientific impossibility for this bomb in this configuration and placement to bring down any 747, a fact well-known to bomb makers, so why would any "Arab" or "Moslem" group now be planning to use another bomb in exactly the same way? They would not of course, which brings us back to the story in the Murdoch newspapers on 15 August 1999. What this story is all about is making the public believe once and for all that a bomb of this sort can bring down a 747, making the forged evidence more "believable" at the trial starting in May 2000. And if the real culprits feel obliged to blow up another Boeing 747 with another 259 passengers on board to prove their point, rest assured they will do so. This time, however, they will blame the atrocity on new Arab Moslem "Terrorist of the Month" Osama Bin Laden.
         The psychological cues for this second atrocity are already in place, proved by the Murdoch story in which reporter Peter Rose claims "British and US airlines are on top security alert after warnings that Islamic fanatics are planning a Lockerbie-style bombing." Rose continues "Airline staff have been ordered to be extra vigilant in checking passengers and luggage. The warning, from the Department of Transport, which follows a tip-off from US Intelligence, specifically mentions Samsonite suitcases." The key psychological cues are "Lockerbie", "Samsonite" and "Suitcases". When the next 747 explodes in mid-air, US Intelligence will provide a helpful tip-off that this was another "Lockerbie" style atrocity, caused by a bomb in a "Samsonite Suitcase" planted by Islamic fanatics, most probably led by Osama Bin Laden. Properly outraged, you the gullible public will then cheer on the prosecutors at the Libyan trial.
         For terrorism buffs, here is why the forged American "evidence" against the two Libyans is a lie, and why the 312 gram Semtex-H bomb could not have brought down Pan Am 103 over Lockerbie, or anywhere else. The "evidence" claims that the bomb was constructed in such a way that its barometric switch activated when the aircraft climbed through 10,000 feet altitude, in turn starting the bomb timer. When the timer reached zero the bomb detonated, blowing a large hole in the forward left pressure hull of the Jumbo, causing it to break up and fall to earth at Lockerbie.
         The "barometric switch" could never have activated at 10,000 feet on the Air Malta Boeing 737 between Malta and Frankfurt, or on the Pan Am feeder Boeing 727 between Frankfurt and London, or on the Boeing 747 designated Pan Am Flight 103 out of London for New York. The reason it could not is disarmingly simple, and known to every flight engineer on earth. The passenger cabin and baggage holds on all Boeing 727s, 737s and 747s are located inside the pressurised hull, which is designed to protect passengers from the rarefied atmosphere at its cruising altitude of about 36,000 feet, where oxygen is insufficient to sustain life. So when the aircraft takes off and climbs towards its cruising altitude, the actual altitude inside the passenger cabin and baggage holds remains much lower, well below 8,000 feet at all times, and normally below 6,000 feet, to ensure you have enough oxygen to breathe comfortably while you sip a brandy or two and watch the in-flight movie.
         This is an incredibly important fact, because the inventors of the forged "evidence" have stated emphatically in writing that the barometric switch was designed to start the bomb timer when the aircraft climbed through 10,000 feet. Evidently (and fortunately) the CIA knows nothing about flight engineering, because the fictional barometric switch was in the pressurised baggage hold and thus never exceeded a maximum altitude of 8,000 feet at any point in the flight. Unless the switch reaches 10,000 feet it cannot activate itself, cannot start a timer, and therefore cannot detonate any bomb. By failing to hire an expert to forge its bomb "evidence" the culprits shot themselves through both feet, and hopefully as a result the two innocent Libyans will be set free next year.
         The timer itself is of little consequence, because anyone can drop charred timer parts at the scene of a crash two years after the event, and evidence to hand suggests the CIA did exactly that. Its main reason for doing so was that sources in Europe had discovered the Libyans had bought a limited series of timers from a Swiss manufacturer, so the CIA used this data to point the finger at Tripoli. Unluckily for the devious CIA,  the Swiss manufacturer has recently pointed out that the printed circuit boards for the timers were different colours, and the timer in question was never sold to the Libyans at all.
         Now to the blast potential of the fake bomb in the Toshiba Bombeat radio, though this will only be covered in outline because detailed information is apparently being reserved for trial defence. It is claimed that 312 grams of Semtex-H explosive in a plastic radio was encased in woollen clothes, which in turn were encased in a Samsonite suitcase. The suitcase was then loaded into a sealed alloy baggage bin which was loaded into the forward baggage hold of Pan Am 103, separated from the immensely strong pressure hull of the aircraft by an air gap of several inches.
         The shock wave from any explosion takes the line of least resistance, decreasing in pressure (p.s.i.) the further it travels from the epicentre of the blast. If this relatively puny bomb had been detonated as claimed on Pan Am 103, the initial shock wave would have been partly deflected and decreased by expansion around the clothes inside the Samsonite, then deflected and decreased still further as it expanded to fill the sealed alloy baggage bin. If the shock wave was sufficient to burst the baggage bin, huge expansion would then take place in the very large forward baggage hold zone, before coming into direct contact with the immensely strong outer pressure hull. Rough calculations show the pressure of the shock wave at this stage would have dropped below 3 p.s.i. before contacting the outer pressure hull itself, which is capable of withstanding at least 8 p.s.i. and (reportedly) as much as 12 p.s.i.
         There is only one sure way that 312 grams of Semtex-H can puncture the pressure hull of a Boeing 747, and that is to be placed directly against the hull itself in the form of a "shaped charge". In order to do this the baggage hold lining must first be removed to gain access to the hull, a complicated procedure which can only be carried out in the New York Pan Am engineering bay at John F. Kennedy Airport. Ironically perhaps, Pam Am 103 was wheeled out of this engineering bay only one hour before it flew to London early on 21 December 1988. There were no Libyans present in the engineering bay.
         The CIA case is a total myth, which is starting to fall apart as each long day drags out towards the trial of the two Libyans in May 2000, and as stated earlier some of the western "authorities" are getting very nervous. It must be evident that unless something dramatic is done, public scepticism will grow to the point where the Libyan trial will collapse completely.
         First item on the agenda must surely be that of "proving" the CIA "evidence" is feasible, pointing once again to the strange story peddled by the Murdoch newspapers. Readers might like to consider whether there is any pressing need for their relatives or friends to take an airline flight during the year, especially on a Boeing 747 flying an international route. Travelling by bus might be a little slower but it is likely to be much safer, at least until after the Libyan trial ends.



The Falklands Alternative, click here

After instructing its Ambassador to tell Saddam Hussein that America "Has no problem with your border dispute with Kuwait", the US State Department sat back smugly and waited for the inevitable invasion.The destruction of Iraq would enable religious fanatics in New York to expand eastwards towards a "Greater Israel". It would also kick oil prices high enough to start exploitation of Falklands oil. Two birds with one stone.  
The Port Arthur Massacre, Tasmania, click here

.On Sunday 28 April 1996, an unknown professional combat shooter opened fire in the Broad Arrow Cafe at Port Arthur in Tasmania. In less than a minute 20 people lay dead, 19 of them killed with single shots to the head, fired from the right hip of the fast-moving shooter. The awesome display of combat marksmanship was blamed on intellectually impaired Martin Bryant, who was held in illegal strict solitary confinement for more than 120 days, until he was "ready" to plead guilty. There was no trial. 
Electronically Hijacking the WTC Attack Aircraft, click here

Forget the media hype about "Arab hijackers" because there were none on board any of the aircraft on 11 September. Outrageous, you say? Not at all. The four aircraft used in the attacks on the World Trade Center and Pentagon, were hijacked electronically from the ground, and flown into their respective targets without the consent of the pilots, who were rendered helpless by the advanced technology used. 
Crash of Pan Am 103 at Lockerbie, click here

When Pan Am 103 exploded at Lockerbie, the media hyped up "Moslem Terrorists". First the Syrians, then the Iranians, and finally the Libyans.  The most obvious suspect should have been the State of Israel, because it alone stood to gain from blackening the reputations of Arab nations.  Despite an earlier known attempt to bomb one of its very own El Al airliners in 1986 for media effect, the probability that the Mossad was also responsible for bombing Pan Am Flight 103, was ruthlessly suppressed.
Vialls Investigations
Exposing Media Disinformation
Misinformation is sometimes accidental,
Disinformation is always intentional


Joe Vialls - Brief History of Web Site

This web site covers three major investigations conducted since 1992. The first is an independent investigation into the murder of policewoman Yvonne Fletcher outside the Libyan Embassy in London during 1984, and links to the CIA and Israeli Mossad. The second is an investigation into the bombing of Pan Am 103 over Lockerbie in Scotland during 1988, and the trial of the two accused Libyans at Camp Zeist (Kamp Zeist) in Holland. Once again with links to the CIA and Mossad. The third and last investigation takes a long hard look at the Port Arthur Massacre in Tasmania, Australia, a crime for which intellectually impaired Martin Bryant was convicted without trial.  A brief overview of each individual case follows.

The Murder of Policewoman Yvonne Fletcher, copyright Joe Vialls

On the 17th April 1984, policewoman Yvonne Fletcher was shot in the back while on crowd control duties outside the Libyan Embassy in London. In the ensuing media frenzy, Libya was accused of ordering her death, and its diplomatic personnel were expelled from the Embassy and from London. The subsequent coronial inquest into her death was rushed and lacking in detail. On closer examination of the available scientific evidence, it became very clear that the shot which killed Yvonne Fletcher could not have been fired from the Libyan Embassy, but only from Enserch House a few doors away. In-depth investigation proved links between this building in 1984 directly to the CIA, and indirectly to the Israeli Mossad. Exactly why these two agencies ordered the execution of an unarmed policewoman doing her duty on the streets of London is unclear, but general motivation seems obvious. Israel in particular has always viewed Libya as an enemy, and the murder of Yvonne Fletcher completely undermined Libya's standing in the international community. In addition, economic sanctions rapidly imposed on Libya caused severe damage to its defence, health and other internal structures.

Lockerbie and the Bombing of Pan Am 103, copyright Joe Vialls

On 21st December 1988, Pan Am 103 exploded overhead Dean's Cross in the English Lake District, its 500 m.p.h. forward speed and inertia ensuring the actual crash site was far to the north at Lockerbie in Scotland. Initially the finger of suspicion was pointed at Syria and Iran, then very conveniently at Libya during the Gulf War, when America badly needed Syrian support for its attacks on Baghdad. There was never any proof of Libyan complicity in the bombing or subsequent crash of Pan Am 103 at Lockerbie, except for evidence which was clearly forged, and predictable media hysteria. The trial of the two Libyan suspects has started at Camp Zeist (Kamp Zeist) in Holland, and poses a huge problem for western nations, America in particular. At the time the allegations were aired it seemed certain Libya would not give up its citizens for international trial. But now Libya has given up its citizens for trial under Scottish Law, America will have to publicly explain why the accusations were made in the first place, and why Libya was so blatantly accused of terrorism. Due to tough rules of evidence this may well prove impossible.

The Port Arthur Massacre, Copyright Joe Vialls

Just after noon on 28th April 1996, an unknown marksman opened fire on diners in the Broad Arrow Cafe at Port Arthur in Australia. In less than 20 minutes at this and five other crime scenes, the marksman killed 35, injured 22, and crippled two cars with only 64 shots. Nineteen of the first twenty dead in the Broad Arrow Cafe died from single shots to the head, all fired by the unknown marksman from his right hip. This staggering display of marksmanship was blamed on left-handed and intellectually impaired Martin Bryant, who had no shooting experience, or military training of any kind. From the time of his arrest, remand prisoner Martin Bryant was illegally held in strict solitary confinement and denied access to media of any kind until his interrogation on 4th July 1996. When he refused to admit to the Port Arthur Massacre at interrogation, he was once more placed back in illegal solitary confinement. Eventually in desperation during November 1996, Martin Bryant pleaded "guilty" 72 times, thereby allowing the authorities to avoid a humiliating trial at which they could present no hard evidence of guilt. Intellectually impaired Bryant was first convicted by a hysterical media pack, then forced to plead guilty by prison officials illegally enforcing strict solitary confinement.

Joe Vialls is a Private Investigator with thirty years direct experience
of international military and multinational operations

Contact:

Joe Vialls
45 Merlin Drive
Carine
Western Australia 6020


Crash of Pan Am 103 at Lockerbie, click here

When Pan Am 103 exploded at Lockerbie, the media hyped up "Moslem Terrorists". First the Syrians, then the Iranians, and finally the Libyans.  The most obvious suspect should have been the State of Israel, because it alone stood to gain from blackening the reputations of Arab nations.  Despite an earlier known attempt to bomb one of its very own El Al airliners in 1986 for media effect, the probability that the Mossad was also responsible for bombing Pan Am Flight 103, was ruthlessly suppressed.
Electronically Hijacking the WTC Attack Aircraft, click here

Forget the media hype about "Arab hijackers" because there were none on board any of the aircraft on 11 September. Outrageous, you say? Not at all. The four aircraft used in the attacks on the World Trade Center and Pentagon, were hijacked electronically from the ground, and flown into their respective targets without the consent of the pilots, who were rendered helpless by the advanced technology used. 
The Port Arthur Massacre, Tasmania, click here

.On Sunday 28 April 1996, an unknown professional combat shooter opened fire in the Broad Arrow Cafe at Port Arthur in Tasmania. In less than a minute 20 people lay dead, 19 of them killed with single shots to the head, fired from the right hip of the fast-moving shooter. The awesome display of combat marksmanship was blamed on intellectually impaired Martin Bryant, who was held in illegal strict solitary confinement for more than 120 days, until he was "ready" to plead guilty. There was no trial. 
The Falklands Alternative, click here

After instructing its Ambassador to tell Saddam Hussein that America "Has no problem with your border dispute with Kuwait", the US State Department sat back smugly and waited for the inevitable invasion.The destruction of Iraq would enable religious fanatics in New York to expand eastwards towards a "Greater Israel". It would also kick oil prices high enough to start exploitation of Falklands oil. Two birds with one stone.  
Cancer and Vitamin B17 Laetrile, click here

For nearly thirty years the media blasted you with scare stories about the "deadly" cyanide content in Vitamin B17 Laetrile, warning that anyone daring to eat apricot kernals would fall dead on the floor in seconds. This is simply not true, and B17 remains one of the most potent controls for cancer anywhere in the world. Recently medical researchers in London "discovered" that the cyanide found in B17 can kill tumours, though of course no-one dared mention apricots. The researchers used cyanide derived from the Cassava root instead, which as you will see here contains exactly the same active cyanide ingredients as apricots!

IS CANCER MERELY A VITAMIN DEFICIENCY DISEASE?
Copyright Joe Vialls, edited 20 Jan 2001, All Rights reserved

         Though many alternative treatments for cancer have demonstrated success over the years, there has been too little discussion on possible ways of preventing or suppressing cancer before it has a chance to attack, or controlling it effectively after it strikes. There is also the question of why we have become increasingly vulnerable to every form of cancer as each year goes by. Is there a direct link between easily-researched and steadily increasing quantities of chemicals and additives in our food and water supplies, or has the removal of certain intrinsic factors from our refined western diets been more to blame?  Specifically where vulnerability is concerned, there is compelling evidence that the removal of vitamin B17 from our diets has played the greatest single role.
         Decades ago two books provided vital clues to one possible cause for the alarming increasing incidence of all forms of cancer. Though aimed at widely different readerships, both books looked at possible connections  between vitamin deficiency and disease.  "World Without Cancer" by Edward Griffin of California examines the growing conviction that vitamin B17 appears capable of preventing the onset of all forms of cancer, while the second book "Eat Fat and Grow Slim" by Richard MacKarness M.D., proposes a high fat diet for those tired of trying the 'lettuce leaf' approach to weight loss.
         Initially it is difficult to see any connection between these two widely differing subjects, but connection there is. In their individual ways both books point towards two entirely different groups of people (one vegetarian and the other carnivorous) who suffer no cancers or heart disease at all while consuming local food in their native environments. This is a critical point, for without such a linkage it is far too easy for established medical researchers to lead the public astray with billions of dollars worth of research aimed at promoting radiation and chemotherapy treatments. During 1950 after many years of research a dedicated biochemist by the name of Dr. Ernst T. Krebs, Jr. isolated a new vitamin that he numbered B17 and called 'Laetrile'. As the years rolled by, thousands became convinced that Krebs had finally found the complete control for all cancers, a conviction shared by even more people today. Back in 1950 Ernst Krebs had no idea of the hornet's nest he was about to stir up.
         Unable to patent B17 or claim exclusive rights to the vitamin, the pharmaceutical multinationals launched a massive propaganda attack of unprecedented viciousness against Laetrile, despite the fact that hard proof of its efficiency in controlling cancer now surrounds us. How is it any of us gets cancer in the first place - through exposure to cigarette smoking, intense sunlight or perhaps the effect of toxic food additives? Dr. Krebs thinks not. All of his hard biochemical evidence points to the fact that cancer is a simple deficiency disease of vitamin B17, long ago removed from our highly refined western diets. Krebs postulates that the so-called 'carcinogens' are merely stress triggers which  finally expose the B17 deficiency with devastating effect.  The credibility of Krebs' claim is best illustrated by the vitamin C deficiency disease known as 'scurvy'. As with cancer there is no advance warning of scurvy; no tell-tale signs that the body is running low on vitamin C reserves. One minute the patient is a healthy person and the next an invalid. Recovery from scurvy is equally dramatic. Within days (sometimes hours) of high-dose vitamin C treatment the scurvy vanishes, reappearing only if vitamin C reserves once more drop below a critical (but undocumented) level.  Thus if Ernst Krebs is right, alternative treatments such as Essiac, Oxygen, and electromagnetic therapies are continually fighting an uphill battle. The simple replacement of the 'lost' Vitamin B17 in our diets might contribute to an increased success rate for other alternative treatments, or replace them completely.
         The proof Dr. Krebs has presented over the years to support his claim of 'lost' B17 in our diets is impressive. Centuries ago we used to eat millet and linseed bread rich in B17, but now we chew our way through wheat bread which has none at all. For generations our grandmothers used to carefully crush the seeds of plums, greengages, cherries, apples, apricots and other members of the botanical family Rosaceae, and diligently mix the kernels with their home made jams and preserves.  Grandma probably didn't know why she was doing it, but the kernels of all these fruits are some of the most potent sources of B17 in the world.  In the tropics, huge quantities of B17 are found in bitter cassava, also known as tropical manioc. Research has proved that a Himalayan tribe known as the 'Hunza' never contract cancer or suffer from heart disease if they stick to their native diet which is exceptionally high in both apricots and millet. However, once exposed to western diets the Hunza become as vulnerable as the rest of us. Because the  Hunza eat very little meat this might encourage large numbers of the vegetarian community to pat each other eagerly on the back whilst proclaiming: "See, we were right!"
         Alas, such jubilation would be sadly misplaced.  In a bid to find a diet acceptable to those not wishing to nibble one lettuce leaf a day, Richard MacKarness made a detailed study of Eskimos living on the polar ice, and American Indians eating traditional diets.  In their natural environments both groups are mostly carnivorous, eating wild game including Elk and Caribou, supplemented only by  wild berries when available in season. The main point MacKarness makes in his book "Eat Fat and Grow Slim" is that there is no such thing as obesity among these people: an interesting fact in its own right as they regularly gorge themselves on saturated animal fats at least twice a day. Where things get decidedly more interesting is his proof that Eskimos and Indians living in their natural environments and eating traditional foods, NEVER contract cancer or suffer from heart  complaints: exactly the same as the Hunza people in the Himalayas, despite the Eskimos and American Indians being carnivores rather than vegetarians. Careful investigation reveals the most likely common factor to be vitamin B17. The caribou which form a large part of the staple diet of both groups graze predominantly on arrow grass containing up to 15,000 mg per kilo nitriloside, the primary source of B17.  The salmon berries dried and eaten by Eskimos and Indians alike also contain huge quantities of vitamin B17. So in these widely differing communities vegetarians and carnivores alike can both remain perfectly healthy. This is of particular importance to those who are environmentally unable to take up a vegetarian diet by choice.  Such a diet would be well nigh impossible on the polar ice caps or in arid deserts.
         Unfortunately for most 'civilized' western cultures, grasses and other foodstuffs now used to feed domestic animals intended for human consumption rarely contain more than a trace of nitriloside, though they did until botanists and biochemists started  to genetically alter our plant life. In turn this means our secondary source of vitamin B17 (through the meat food-chain) is fast drying up. Where The Hunza or Eskimaux get an average individual ration of between 250 and 3,000 milligrams of vitamin B17 every day, European folk eating 'healthy' modern foods receive barely 2 milligrams.  The implications of these finding are staggering of course. If we managed to control scurvy centuries ago, how is it we cannot do the same for cancer today?  The fact is we probably could if our respective governments would allow it.  Unfortunately most: governments have buckled under the pressure exerted by the pharmaceutical multinationals, the American Food & Drug Administration, and the American Medical Association. All three have mounted highly successful 'scare' campaigns based on the fact that vitamin B17 contains quantities of 'deadly' cyanide; conveniently forgetting that vitamin B12 also contains large quantities of cyanide but is freely available in health food shops world-wide.
         Dr. Kreb's B17 Laetrile was derived from apricot kernels and then synthesised into crystalline form using his own unique process.  Suddenly the American FDA bombarded the media with a  story about an unfortunate couple who had poisoned themselves by eating raw apricot kernels in San Francisco. The story made headline news across the U.S.A. although several suspicious ;journalists never managed to establish the identity of the unfortunate couple, despite many determined attempts.  But the multinational pharmaceutical/FDA boot had been put in with a vengeance. From that point onwards eating apricot kernels or B17 Laetrile became synonymous with committing suicide. Back in the fifties Dr. Ernst Krebs proved beyond doubt that B17 was completely harmless to humans in the most convincing way possible. After testing the vitamin on animals, he filled a large hypodermic with a mega-dose of concentrated Laetrile which he then injected into his own arm! Drastic perhaps, but the adventurous Dr. Krebs is still alive and well today.
         The vitamin is harmless to healthy tissue for a very simple  reason:  each molecule of B17 contains one unit of cyanide, one unit of benzaldehyde and two of glucose (sugar) tightly locked together. In order for the cyanide to become dangerous it is first necessary to 'unlock' the molecule to release it, a trick that can only be performed by an enzyme called beta-glucosidase, which is present all over the human body in minute quantities, but in truly vast quantities (up to 100 times as high) at only one place: the site of a malignant cancer tumour. Thus the cyanide is unlocked only at the cancer site with drastic results, which become utterly devastating to the cancer cells because the benzaldehyde unit unlocks at the same time.  Benzaldehyde is a deadly poison in its own right, which then acts synergistically with the cyanide to produce a poison 100 times more deadly than either in isolation. The combined effect on the cancer cells is best left to the imagination. But what about danger to the rest of the body's cells? Another enzyme, rhodanese, always present in far larger quantities than the unlocking enzyme beta-glucosidase in healthy tissues, has the easy ability to completely break down both cyanide and benzaldehyde into beneficial body products. Predictably perhaps, malignant cancer cells contain no rhodanese at all, leaving them completely at the mercy of the two deadly poisons.
         Generations ago our agricultural experts knew of the 'trigger' effect of beta-glucosidase i.e. its ability to unlock the cyanide unit in the B17 molecule, but there appeared to be a considerable amount of confusion about how to approach the problem. The simplistic solution seemed to be that of labeling all plants containing the B17 molecule "poisonous", then genetically modifying them to remove the nitriloside content completely for the safety of the animals. One classic example of this misguided approach was a 1940s case where Australian sheep were occasionally dying from an excess of cyanide derived from white clover, known to contain B17. Without giving a thought to why most of the sheep eating the same clover stayed alive,  botanists promptly bred the nitriloside content out of the white clover. In reality the sheep that died were the few who wandered away from the clover to eat a tasty fuschia plant which contained a very high concentration of the unlocking enzyme beta-glucosidase, which reacted immediately in the sheeps' stomachs and caused death. If the botanists had neutralised a few fuschias instead of millions of tons of white clover, there would be significantly more vitamin B17 available today for humans to ingest through the meat food-chain.
         For better or worse vast quantities of vitamin B17 have been removed from western foods, and society is now faced with cancers at an unprecedented level. Even if we allow that a deficiency of B17 might be the most likely culprit for the sudden appearance of such a condition, there is still the question of what happens next and how that cancer develops to the life-threatening stage. In "World Without Cancer", Griffin explains the trophoblastic theory of cancer proposed by Professor John Beard of Edinburgh University, who claims certain pre-embryonic cells in pregnancy differ in no discernible way from highly malignant cancer cells. Griffin notes: ' The  trophoblast  in pregnancy  indeed  does  exhibit  all the   classical characteristics of cancer. It spreads and multiplies rapidly as it eats its way into the uterus wall preparing a   place where the embryo can attach itself for maternal protection and nourishment.' 
         The trophoblast is formed in a chain reaction by another cell which Griffin simplifies down to the 'total-life' cell, which can evolve into any organ or tissue, or alternatively into a  complete human embryo. When the total-life  cell is triggered into producing trophoblast by contact with the hormone oestrogen, present in both males and females, one of two different things happens: in the case of pregnancy the result is conventional development of a placenta and umbilical cord. If the trophoblast is triggered as part of a healing process however, the result is cancer or, as Edward Griffin cautions: 'To be more accurate, we should say it is cancer if the healing process is not terminated upon completion of its task.' Stunning proof of this claim is readily available. All trophoblast cells produce a unique hormone called the chorionic gonadotrophic (CGH) which is easily detected in urine. Thus if a person is either pregnant or has cancer, a simple CGH pregnancy test should confirm either or both. It does, with a reported accuracy of better than 85%. If the urine sample shows positive it means either normal pregnancy or abnormal malignant cancer. Griffin notes: 'If the patient is a woman, she either is pregnant or has cancer. If he is a man, cancer can be the only cause.'  So why all of the expensive, dangerous biopsies carried to 'detect' cancerous growths? One can only assume that medicare pays doctors a larger fee for biopsies than pregnancy tests. In Australia, two CGH style 'do-it-yourself' pregnancy tests stocked by most pharmacies are 'Discover' and 'Predictor'.
         Physicians reading this article will probably be shaking with self-righteous indignation by this stage, muttering darkly: 'Yes, but where is the PROOF?' Here: Most people have heard of 'spontaneous regression' where  a cancer simply goes away, hopefully never to reappear. Such spontaneous regressions are exceedingly rare and vary from one form of cancer to another. One virulent cancer variety known as testicular  chorionepithelioma has never been known to produce a single spontaneous regression. Perhaps for that precise reason, Dr.  Krebs singled it out for special attention when proving the effectiveness of B17 Laetrile in providing total control for cancers.
         As Edward Griffin recalls, in a banquet speech in San Francisco on November 19, 1967, Dr.  Ernst T. Krebs briefly reviewed six cases of testicular chorionepithelioma.  Then he added: 'Now there is an advantage in not having had prior radiation, because if you have not received prior radiation that has failed, then you cannot enjoy the imagined benefits of the delayed effects of prior radiation. So this boy falls into the category of the "spontaneous regression." And when we look at this scientifically, we know that spontaneous regression occurs in fewer than one in 150,000 cases of cancer. The statistical possibility of spontaneous regression accounting for the complete resolution of six successive cases of testicular chorionepithelioma [All six completely resolved solely by B17 Laetrile - Ed.] is far greater than the statistical improbability of the sun not rising tomorrow morning. '
         Wisely perhaps, Griffin notes that because of the adverse publicity against B17 Laetrile, and because of the difficulties in obtaining the 'banned' substance, most cancer sufferers turn to the vitamin as a last resort, long after they have been burned by radiation therapy, and/or poisoned by chemotherapy.
         When "World Without Cancer" was written in 1974, B17 Laetrile  was freely available in Australia. It is not now. A recent check with the Australian Cancer Foundation and health authorities revealed that nowadays Canberra considers each individual case on its merits, then decides whether the patient should be allowed to import sufficient of the material for his or her own personal use. If he or she manages to jump that hurdle, it is then his or her own responsibility to find a doctor prepared to inject it.  Seemingly the multinational lobbyists managed to get to our politicians before  Dr. Krebs could get to the Australian public.
         Last month Australian nationwide television carried the frightening news that two out of every three Australians can expect to suffer skin cancer at least once during their lifetimes. On the massive evidence provided by Dr. Ernst Krebs, Jr., Edward Griffin and Dr. Richard MacKarness, that figure might be crushed to a tiny percentage if Australians were allowed freedom of choice where B17 Laetrile is concerned.  It is perhaps time for Australians to take a stand on this undeniably lethal issue.


REFERENCES


Australian Veterinary Journal, Franklin and Reid, Volume 100, p92, 1944.


CANCER: Disease of Civilization? An Anthropological and Historical Study, Stefanson, V, Hill & Wang, New York, 1960.


Eat Fat and Grow Slim, MacKarness, R., Fontana, London 1976.


Laetrile Case Histories, Richardson J.A. and P, Bantam, USA.


New Zealand Journal of Science and Technology, Coop & Blakely, February 1949, page 277,  February 1950, page 45.


The Nitrilosides in Plants and Animals, "Nutritional and therapeutic Implications", Krebs E.T, Cancer Book House, Los Angeles, California, USA.  Published by the Cancer Control Society, a non-profit educational Society dedicated to educating the public about nutrition and non-toxic cancer therapies.


World Without Cancer, Griffin, G.E., American Media, PO Box 4646, Westlake Village, California 91359, USA. Price US$9.95 plus air mail postage of US$9.00. Discounts available (up to 65% for bulk orders) for alternative booksellers.


Footnote 1997


         For anyone unable to access synthesised Laetrile in Mexico, the most obvious source of oral vitamin B17 is apricot kernels. Unfortunately the Australian Government has made access to this natural product extremely difficult. Courtesy of the pharmaceutical lobbies, it is now a federal "crime" for health food shops and others to sell apricot kernels to the public. There is an easier way of obtaining B17 not shown in the text, and that is from crushed linseed. The primary source of B17 in linseed is Linimarin rather than Nitriloside found in apricot kernels and Cassava, but the effect is the same if enough is eaten. 
         Readers should note that linimarin B17 exists in the crushed linseed itself, not in the extracted linseed oil.  Cheap "Linseed Cake" sold by all animal feed stores is ideal, though in every case the linseed should be mixed in a high-speed blender or coffee grinder before use. Whole linseed is very hard, passing through the gut unaltered and without releasing its beneficial B17 content into the bloodstream. Once crushed, the linseed meal may be mixed with breakfast cereals or baked in bread.
         This report was first published in "Conscious Living" magazine, issue 27, February 1995, and has since been posted on a number of different internet sites.



"SETTING UP" LIBYA FOR THE LOCKERBIE BOMBING"
Copyright Joe Vialls, January 2000


         Most media hype alleging the two Libyans currently imprisoned in Holland are guilty of the bombing of Pan Am 103 over Lockerbie, can be traced back to a single book. Co-authored in 1993 by Donald Goddard and Lester Coleman III, "Trail Of The Octopus" spawned thousands of stories in the mainstream media. Indeed, without "Trail Of The Octopus" as a guide, it is difficult to see how the media could have generated any meaningful stories at all about Libyan "guilt" over the Lockerbie bombing. At the surface level the book deftly tries to shift part of the blame backs towards Israel's prime enemy Syria, thus embroiling yet another Arab nation in a sea of deceit and half truths. Whoever blew Pan Am 103 up over Lockerbie, the book all but shouts at the reader: "The culprits were not America or the State of Israel". 
         In essence the text is a series of recollections by Lester Coleman III, ghost written and edited by journalist Donald Goddard. Coleman claims to have been a consultant for the American Defense Intelligence Agency (DIA) with strong links to the Drug Enforcement Agency (DEA). If his claims are true, this would of course put Lester Coleman III in the driving seat on matters related to the Middle East, and in particular the bombing of Pan Am 103. But are his claims really true? The media made the fatal mistake of assuming they are, thereby setting up a huge house of cards ready for instant destruction by anyone sceptical enough to ask serious questions. If Lester Coleman III's claims can be proven untrue, then by default every one of the thousands of media stories which followed about Lockerbie are also untrue.
         This report is not about minor grammatical errors made by Goddard, or marginal recall about the spelling of an obscure contact's name by Coleman. It is about proven errors so large you could drive a 10-ton Mack truck through each and every one of them with room to spare.
         By his own account Coleman first arrived in Beirut for the DIA on 25 February 1985. At that time he claims part of his job was to liaise with, and provide intelligence for, the Green Berets [special forces] in place for a hostage rescue mission. In the words of Donald Goddard on p 144: " the main duties of the Asmar cell had been to monitor the Muslim radical groups supported by Iran and Syria, to report on the movements of their leaders - in particular, those of Sheikh Mohammed Hussein Fadellah - and keep track of the Western hostages then being taken in Beirut. After Coleman's arrival as Asmar's control and his communications link with DIA headquarters, this work continued "
         So Coleman specifically claims through Goddard in the book that he [Coleman] had a direct controlling role in the Beirut hostage drama from 25 February 1985. Later in the text he expands on this theme by mentioning hostage names such as Levin, Anderson, Keenan, McCarthy and Waite, but erroneously insists there was no rush about freeing the hostages at that time. Indeed, on pp 182-183 he claims: "We could have gotten the hostages out any damn time we wanted to, but nobody was willing to rock the boat with a rescue operation. We knew where they were. We knew who their guards were. We knew what they had for lunch. We knew when and where they were going to be moved before their guards did. But the DIA wouldn't risk any action based on information that might have been traced back to one of Asmar's people."
         This would have come as an enormous shock during early 1985 to no less a personage than William Casey, Director of the CIA, who at the time had a close working relationship with the heads of both the DIA and DEA. On 16 March 1984 his own CIA head of station in Beirut, William Buckley, was captured and became by far the most important hostage from the American Intelligence Community's point of view. Hizbollah was holding a senior member of the CIA hostage, and [according to Casey] slowly torturing him to death during the exact period Coleman claims that "We could have gotten the hostages out any damn time we wanted to".
         Unfortunately Coleman proves himself ignorant of this pivotal fact, and demonstrates he has no knowledge of the swarms of spooks criss-crossing Beirut, trying just about anything to get Buckley out of there during early 1985. Buckley's name is not mentioned anywhere in the text or index of "Trail of the Octopus" at all. Coleman proves he is also unaware that the Green Berets he "had to liaise with, and provide intelligence for", were in Beirut at that time primarily to try a snatch job on William Buckley.
         According to David Jacobsen, director of the city's American University Hospital, who was taken hostage some weeks before and was sharing a cell with Buckley, the latter finally died from a combination of abuse and pneumonia on his 444th day of captivity, during the evening of 3 June 1985. Hizbollah did not announce Buckley's death until 11 October 1985, but no matter which of the two dates is correct, the period January - June 1985 in Beirut was frantic, because Casey had pulled out all the stops and ordered a rescue mission regardless of cost.
         Predictably perhaps, Jacobsen also appears to have slipped Coleman's memory. Just like William Buckley's, his name is also missing from "Trail Of The Octopus". Because of Buckley's profile, and Casey's directive as Director of Central Intelligence (DCI), it is ridiculous to suggest that Coleman could "forget" to include this critical information in "Trail of the Octopus".
         Over the years I have attempted to investigate this staggering erroneous claim as best I could, because if Coleman was not performing the duties he claimed in Beirut during the time frame claimed in the book, then most of his other claims made in "Trail Of The Octopus" can reasonably be regarded as untrue and deliberately fabricated disinformation.
          Not one of the personnel I have so far contacted at Fort Bragg, North Carolina, remembers liaising with or receiving intelligence from, Lester Coleman III or his alias "Thomas Leavy" in Beirut.
         In late October 1996, Lester Coleman III was badly beaten by four masked men at his hideout in Spain. On arrival back on America on 17 October 1996 in a wheelchair, Coleman was arrested by the U.S. federal authorities, and charged with passport fraud. Hardly the treatment one would expect to be meted out by federal authorities to a bona-fide Defense Intelligence Agency consultant.

PSYCHOLOGICALLY ENGINEERING THE LOCKERBIE TRIAL

         On 15 August 1999 in a story titled "Airlines Fear Bomb Attacks", Murdoch Group newspapers suggested Islamic fanatics are planning another Lockerbie-style bombing sometime during the next three months. It is far more likely that those really responsible for bombing Pan Am 103 are planning a lookalike atrocity, designed to act as a psychological reinforcer during the trial of the two Libyan suspects in late 2000. There is no credible scientific evidence linking these two men to Pan Am Flight 103 or to the village of Lockerbie, but the American Department of State and the Israeli Mossad are growing increasingly desperate for a conviction.
                   In a nutshell the forged evidence against the two Libyans is as follows: The suspects took delivery in Malta of a bomb disguised as a Toshiba Bombeat portable radio, said to contain 312 grams of Semtex-H explosive coupled to a barometric switch and a timer. The suspects allegedly wrapped the bomb inside children's clothes bought in a Malta store, then packed it carefully inside a brown Samsonite suitcase for transit to Pan Am 103. The CIA claims the suitcase was then loaded by the suspects onto Air Malta Flight 180 to Frankfurt, where it was transhipped to a Pan Am 727 feeder flight to London Airport Heathrow. There it was allegedly transhipped again onto "Maid Of The Seas", the 747 which then obligingly exploded overhead Deans Cross in the Lake District, its 500 m.p.h. forward speed and inertia ensuring the debris and bodies finally fell to earth many miles further north at Lockerbie in Scotland.
         As will be explained later, it is a scientific impossibility for this bomb in this configuration and placement to bring down any 747, a fact well-known to bomb makers, so why would any "Arab" or "Moslem" group now be planning to use another bomb in exactly the same way? They would not of course, which brings us back to the story in the Murdoch newspapers on 15 August 1999. What this story is all about is making the public believe once and for all that a bomb of this sort can bring down a 747, making the forged evidence more "believable" at the trial starting in May 2000. And if the real culprits feel obliged to blow up another Boeing 747 with another 259 passengers on board to prove their point, rest assured they will do so. This time, however, they will blame the atrocity on new Arab Moslem "Terrorist of the Month" Osama Bin Laden.
         The psychological cues for this second atrocity are already in place, proved by the Murdoch story in which reporter Peter Rose claims "British and US airlines are on top security alert after warnings that Islamic fanatics are planning a Lockerbie-style bombing." Rose continues "Airline staff have been ordered to be extra vigilant in checking passengers and luggage. The warning, from the Department of Transport, which follows a tip-off from US Intelligence, specifically mentions Samsonite suitcases." The key psychological cues are "Lockerbie", "Samsonite" and "Suitcases". When the next 747 explodes in mid-air, US Intelligence will provide a helpful tip-off that this was another "Lockerbie" style atrocity, caused by a bomb in a "Samsonite Suitcase" planted by Islamic fanatics, most probably led by Osama Bin Laden. Properly outraged, you the gullible public will then cheer on the prosecutors at the Libyan trial.
         For terrorism buffs, here is why the forged American "evidence" against the two Libyans is a lie, and why the 312 gram Semtex-H bomb could not have brought down Pan Am 103 over Lockerbie, or anywhere else. The "evidence" claims that the bomb was constructed in such a way that its barometric switch activated when the aircraft climbed through 10,000 feet altitude, in turn starting the bomb timer. When the timer reached zero the bomb detonated, blowing a large hole in the forward left pressure hull of the Jumbo, causing it to break up and fall to earth at Lockerbie.
         The "barometric switch" could never have activated at 10,000 feet on the Air Malta Boeing 737 between Malta and Frankfurt, or on the Pan Am feeder Boeing 727 between Frankfurt and London, or on the Boeing 747 designated Pan Am Flight 103 out of London for New York. The reason it could not is disarmingly simple, and known to every flight engineer on earth. The passenger cabin and baggage holds on all Boeing 727s, 737s and 747s are located inside the pressurised hull, which is designed to protect passengers from the rarefied atmosphere at its cruising altitude of about 36,000 feet, where oxygen is insufficient to sustain life. So when the aircraft takes off and climbs towards its cruising altitude, the actual altitude inside the passenger cabin and baggage holds remains much lower, well below 8,000 feet at all times, and normally below 6,000 feet, to ensure you have enough oxygen to breathe comfortably while you sip a brandy or two and watch the in-flight movie.
         This is an incredibly important fact, because the inventors of the forged "evidence" have stated emphatically in writing that the barometric switch was designed to start the bomb timer when the aircraft climbed through 10,000 feet. Evidently (and fortunately) the CIA knows nothing about flight engineering, because the fictional barometric switch was in the pressurised baggage hold and thus never exceeded a maximum altitude of 8,000 feet at any point in the flight. Unless the switch reaches 10,000 feet it cannot activate itself, cannot start a timer, and therefore cannot detonate any bomb. By failing to hire an expert to forge its bomb "evidence" the culprits shot themselves through both feet, and hopefully as a result the two innocent Libyans will be set free next year.
         The timer itself is of little consequence, because anyone can drop charred timer parts at the scene of a crash two years after the event, and evidence to hand suggests the CIA did exactly that. Its main reason for doing so was that sources in Europe had discovered the Libyans had bought a limited series of timers from a Swiss manufacturer, so the CIA used this data to point the finger at Tripoli. Unluckily for the devious CIA,  the Swiss manufacturer has recently pointed out that the printed circuit boards for the timers were different colours, and the timer in question was never sold to the Libyans at all.
         Now to the blast potential of the fake bomb in the Toshiba Bombeat radio, though this will only be covered in outline because detailed information is apparently being reserved for trial defence. It is claimed that 312 grams of Semtex-H explosive in a plastic radio was encased in woollen clothes, which in turn were encased in a Samsonite suitcase. The suitcase was then loaded into a sealed alloy baggage bin which was loaded into the forward baggage hold of Pan Am 103, separated from the immensely strong pressure hull of the aircraft by an air gap of several inches.
         The shock wave from any explosion takes the line of least resistance, decreasing in pressure (p.s.i.) the further it travels from the epicentre of the blast. If this relatively puny bomb had been detonated as claimed on Pan Am 103, the initial shock wave would have been partly deflected and decreased by expansion around the clothes inside the Samsonite, then deflected and decreased still further as it expanded to fill the sealed alloy baggage bin. If the shock wave was sufficient to burst the baggage bin, huge expansion would then take place in the very large forward baggage hold zone, before coming into direct contact with the immensely strong outer pressure hull. Rough calculations show the pressure of the shock wave at this stage would have dropped below 3 p.s.i. before contacting the outer pressure hull itself, which is capable of withstanding at least 8 p.s.i. and (reportedly) as much as 12 p.s.i.
         There is only one sure way that 312 grams of Semtex-H can puncture the pressure hull of a Boeing 747, and that is to be placed directly against the hull itself in the form of a "shaped charge". In order to do this the baggage hold lining must first be removed to gain access to the hull, a complicated procedure which can only be carried out in the New York Pan Am engineering bay at John F. Kennedy Airport. Ironically perhaps, Pam Am 103 was wheeled out of this engineering bay only one hour before it flew to London early on 21 December 1988. There were no Libyans present in the engineering bay.
         The CIA case is a total myth, which is starting to fall apart as each long day drags out towards the trial of the two Libyans in May 2000, and as stated earlier some of the western "authorities" are getting very nervous. It must be evident that unless something dramatic is done, public scepticism will grow to the point where the Libyan trial will collapse completely.
         First item on the agenda must surely be that of "proving" the CIA "evidence" is feasible, pointing once again to the strange story peddled by the Murdoch newspapers. Readers might like to consider whether there is any pressing need for their relatives or friends to take an airline flight during the year, especially on a Boeing 747 flying an international route. Travelling by bus might be a little slower but it is likely to be much safer, at least until after the Libyan trial ends.



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